Thursday, September 30, 2010

Tyler Clementi, eighteen, a freshman at Rutgers University in the United States, jumped off the George Washington Bridge last Wednesday after roommates broadcast his same-sex encounters online.

Clementi shared a room with Dhraun Ravi, in Davidson Hall on Rutgers’ Piscataway campus.

On September 19, Dhraun Ravi is alleged to have secretly activated a webcam in their room, then retreated down the hall to the room of his childhood friend, Molly Wei, where they used Skype to connect to the live video feed of Clementi having sex. Ravi tweeted, “Roommate asked for the room till midnight. I went into molly’s room and turned on my Web cam. I saw him making out with a dude. Yay”.

Clementi reported the incident to the university, and that information has been passed to the authorities, according to Gregory Blimling, university vice president for student affairs.

Ravi attempted to repeat his actions on the following Wednesday, after seeing his roommate kissing a teenaged boy. Writing on Facebook he said, “Anyone with iChat, I dare you to video chat me between the hours of 9:30 and 12. Yes, it’s happening again.”

The following Wednesday, at around 4 p.m., fellow freshman Lauren Garcia saw Clementi leaving the dormitory. “He had his iPod in his ear, and just had this blank stare on his face,”, she said. Clementi drove one hour along the New Jersey Turnpike, abandoning his car on the Jersey side. Writing a final message on his own Facebook page saying, “Jumping off the gw bridge sorry”, he left a wallet containing his driver’s licence and Rutgers ID card, and his mobile phone on the roadside. According to witnesses, he scaled the railings, crouched down, and jumped at approximately 8:50 p.m— about eight minutes after his Facebook posting. Those details were relayed to press anonymously by a law enforcement official.

One of the anonymous witnesses, a 45-year-old from West New York, said “It was so quick, as soon as he saw us, he went. He didn’t give us an opportunity to do anything.”

New York Police Department’s Harbour Unit recovered a body of a young man yesterday, in the Hudson River to the North of the bridge, but it has not yet been positively identified.

Ravi and Wei, both eighteen, were charged with invasion of privacy. Wei voluntarily surrendered to police Monday, and was released on her own recognizance; Ravi surrendered the following day, and has been released on $25,000 bail. All three students started their courses in August.

The crime of “transmitting sexual images” is a 3^rd degree felony, carrying a maximum prison sentence of five years.

Clementi, from Ridgewood, New Jersey, played the violin, and was a member of the Ridgewood High School chamber orchestra. A student from the same hall described him as quiet and shy. His parents were apparently unaware of his sexuality; Robert Righthand, a friend of Clementi since grade school, said “he had it in reserve for a very long time […] You never thought he was depressed. You just thought he was quiet. He wasn’t the person to open up to a lot of people.”

A gathering of around 100 people held a vigil on Wednesday, chanting slogans such as “We’re here, we’re queer, we’re not going home.”

The Parents, Families and Friends of Lesbians and Gays organization announced a “call to action” today. Stephen Goldstein, of LGBT advocacy organization Garden State Equality, said, “[Clementi,] by all accounts, was brilliant, talented and kind […] we are sickened that anyone in our society, such as the students allegedly responsible […] might consider destroying others’ lives as a sport.”

On Wednesday prior to the incident, the university launched “Project Civility”, a two-year campaign encouraging students to be thougtful of others. The University president, Richard McCormick, issued a letter stating that “If the charges are true, these actions gravely violate the university’s standards of decency and humanity.”

Friday, November 6, 2009

A 1965 Volkswagen minibus that was stolen in 1974 has been recovered by customs agents in Los Angeles. The vintage minibus was in pristine condition, valued at $25,000, and was found during a routine inspection of a shipping container scheduled for departure to The Netherlands. A routine computer database search on its vehicle identification number flagged it as having been stolen from a vehicle upholstery shop in Spokane, Washington on July 12, 1974. A custom restoration business in Arizona was attempting to deliver it to overseas clients last month when authorities intercepted the vehicle.

The theft appeared on the National Insurance Crime Bureau database, which is used by border authorities and contains all stolen vehicle records. Most police databases remove unsolved vehicle thefts after five years.

The California Highway Patrol does not suspect the restorer of wrongdoing, according to investigating officer Mike Maleta. Possession of the vehicle apparently changed several times. Police in Spokane have not yet located the rightful owner, whose identity has not been released to the press. Maleta hopes that a trail of registration documents and interviews will uncover the thief.

The Allstate insurance company paid $2500 shortly after the theft occurred and wants to take possession of the vintage minibus. Allstate spokeswoman Megan Brunet expects that after the necessary paperwork is processed the firm will sell it at auction.

Saturday, August 6, 2005

Robin Cook, the former British foreign secretary and current Member of Parliament, has died of a suspected heart attack after collapsing on a walk with his wife on Ben Stack hill, Scotland. Cook, aged 59, was flown to a hospital in Inverness by a rescue service helicopter half an hour after collapsing.

A spokesperson for NHS Highland said that Mr. Cook arrived at hospital 90 minutes after his collapse, and was reported dead five minutes later. A postmortem examination has concluded that he died from hypertensive heart disease.

Mr. Cook was buried on August 12 at St Giles Cathedral in Edinburgh in a service attended by close family, friends and political figures from across the world. British Prime Minister Tony Blair however came under attack from Mr. Cooks friend and racing pundit John McCririck for his absence from the service.

Robin Cook served as Foreign Secretary under New Labour and was a critic of the war in Iraq. He resigned as Leader of the House of Commons in 2003 in protest against Prime Minister Blair’s foreign policies.

Saturday, August 20, 2011

Flight Lieutenant Jon “Eggman” Egging, 33, of the RAF Aerobatic Team (The Red Arrows) has been killed in a crash, shortly after a display in Bournemouth, England. This is the first fatality involving the team for 23 years.

The BAE Hawk jet aircraft crashed at 13:50 BST on the banks of river Stour, near Throop village. A witness, Shaun Spencer-Perkins, said the aircraft “bounced across the field [and] … across the river. Members of the public jumped into the water to search for the cockpit.” Bournemouth Airport closed briefly, following the incident, but has since reopened and is operating normally.

Emergency services were called to the scene, but Egging, who was reportedly thrown clear of the cockpit during the crash, was pronounced dead at the scene, local police announced. He joined the team in autumn 2010 and is the first active member of the team to be killed since 1988. The Ministry of Defence is investigating the cause of the accident.

Egging is believed to have been attempting to avoid nearby houses before the crash. Local residents, who were attempting to rescue the pilot, found an empty ejector seat and parachute at the scene, and believed the pilot to be already dead when they found him.

So far, there is no official explanation for the accident. Amateur footage of the accident shows the aircraft making a tight turn as part of a fan-out manoeuvre, along with other aircraft from the team. Egging’s aircraft then heads towards the horizon, with no apparent radical course change or additional manoeuvre, before disappearing out of sight behind a building. All the aircraft leave a white trail of smoke behind the aircraft, as is a routine part of the Red Arrows display.

Thursday, July 17, 2008

A man shot and killed a Broward County Sheriff’s Office police dog in Miami Beach after a two-county chase in Florida early Thursday morning.

The shooter, Delvin Lewis, 27, was firing shots at his girlfriend during a domestic dispute in Oakland Park. When police arrived, the suspect got into his car and fled, engaging roughly 20 cruisers in a 30-mile chase which ended near Mount Sinai Medical Center’s emergency room in Miami Beach. An exchange of gunfire followed in which Lewis reportedly killed the dog. ER doctors tried to save the animal but it was too late.

The shooter was also struck and has been taken to Jackson Memorial Hospital where he remains with no life threatening injuries.

According to a Mount Sinai spokeswoman hospital facilities are now accessible after the authorities locked down the area for three hours following the shooting.

Lewis has been arrested 26 times by Florida authorities including one in July 2003 in which he injured a police dog striking him repeatedly on the head with a cellphone.The suspect has been charged with aggravated assault with a motor vehicle on a law enforcement officer, aggravated fleeing and eluding, resisting an officer with violence and principal in the death of a police dog.

Hitting a police dog is a third-degree felony punishable by up to five years in jail, while killing a police officer is a capital offense which can lead to the death penalty.

Miami-Dade Police Department is handling the investigation which involves Broward Sheriff Office and Miami Beach police.

Lewis has been in and out of jail since 2000 and had been arrested for domestic battery in 2000 and 2004, when he beat a pregnant woman.

The dog, whose name was Oozi, was a 7-1/2 years old Belgian Malinois assigned to BSO’s Cooper City district. He was trained in narcotics and helped in hundreds of arrests in his career, including 35 this year.

Oozi and his partner, Deputy Gerald Wengert, were named BSO’s Employees of the month in May for their role in the apprehension of three burglary suspects.

Saturday, October 1, 2011

Last week, Nature Genetics carried twin studies into the genetics of bipolar disorder and schizophrenia. This special report examines the month’s research into the illnesses in detail, with Wikinews obtaining comment from experts based in Australia, the United States, and the United Kingdom ahead of the U.S. Mental Illness Awareness Week, which starts tomorrow.

Eleven genetic regions were identified; seven of these were for schizophrenia and five of those were hitherto undiscovered. The parallel studies, conducted separately, examined more than 50,000 people worldwide and identified two genetic loci associated with both diseases.

Little is known about the two illnesses, each of which affects around 1% of people and is treated with strong medication. Bipolar sufferers experience extremes of mood – depression and mania, hence the previous name “manic depression” for the illness. Schizophrenia is associated with hearing voices, chaotic thoughts, and paranoia. There is no known cure.

The latest research examined both the healthy and the afflicted, using computers to scan genomes. Inheritance was thought to be a factor from prior knowledge of the diseases as a familial trait, but the original desire had been to isolate a single faulty gene. Instead it has become apparent that the genetic factors are many; in the case of schizophrenia, at most around 30% of the genetic components are thought to have been identified.

The University of Chicago’s Pablo Gejman, a lead researcher on the schizophrenia study, explained to Wikinews in a telephone interview from Buenos Aires, Argentina that “One of the goals of genetic research is to find druggable targets” – to “find treatments at the root of the problem”.

Whilst noting that there is no guarantee the genetic code identified is druggable, Gejman named calcium-activated neurochemical channels in the brain as candidates for new drugs. The channels were linked to schizophrenia in the study.

Gejman explained that a genetic locus called mir137 “suggests an abnormality of gene regulation.” The diseases are so poorly understood that it is uncertain if they are in fact two components of a single spectrum, or even each comprised of multiple illnesses.

The new and “provocative data” gathered showed the significant loci identified were “not part of the pre-existent hypothesis.” Calling this “interesting”, Gejman added that the team found no evidence that dopamine receptors are involved; current drug treatments target dopamine receptors. The findings are “not related to anything we thought we knew [about schizophrenia],” he told our correspondent.

Quizzed about the possibility variations in the genetic factors involved in expressing the diseases explained the variation seen in symptoms, Gejman was uncertain. “We will have the answer, probably, only when we sequence the whole [human] genome.” He notes that the relationship between genotype and phenotype is unclear, and that “We know very little of the genetic architecture of schizophrenia and” other disorders.At the time the results were published, participating scientist Professor Rodney Scott from the University of Newcastle in Australia said “The strength of this research is in the numbers. The findings are robust and give us a lot of statistical power to identify the genetic determinants of schizophrenia.” Scott told Wikinews that “If any single centre tried to undertake such a study, it would require millions of pounds. Since it was a collection of data from across the world the costs were spread. In this era of financial difficulty it will become increasingly difficult to secure funding for this type of project even though the pay-offs will be significant.”

Gejman expressed similar sentiment. “The research budget is not growing, which makes [funding] difficult,” he said, though he felt the cost “is not prohibitive because of the benefits.” “I think that it was money well invested” and “very well spent for the future,” he said, adding that organisations in Europe and the US were aware of the importance of such research.

Gejman also agreed on reliability – the study is “Very reliable because of the sample size; that should provide robust results… [we] have worked with a much larger sample than before.” Scott told us it was “a highly reliable study” that has the potential to lead to new treatments “in the long run”.

Another point was the two genetic loci identified as common to both – how much support do they lend to the notion the diseases are linked? “Until more information is available it is really only suggestive,” says Scott. “Strong enough to say there may be potentially a common pathway that bifurcates to give rise to two diseases.”

“It is an excellent demonstration,” said Gejman “because you have the same chains that are common to both disorders, in fact not just the same chains but also the same alleles.” He stressed uncertainty in how strong the relationship was, however.

Scott said examining how the variation of genetic factors may translate into varied symptoms being expressed “certainly is a good target for future research”; “It is not known how many genetic factors contribute to either of these diseases but it is likely that not all are necessary to trigger disease.” “New questions will always arise from any major study,” he told our reporter. “Certainly, new questions about bipolar and schizophrenia are now able to be formulated on the basis of the results presented in the two reports.”

These weren’t the only studies to look at the two diseases together in September. The British Medical Journal carried research by a team from the University of Oxford and King’s College London that examined mortality rates in England for schizophrenia and bipolar sufferers. They found both groups continued to suffer higher mortality rates than the general population – whilst these included suicides, three quarters of deaths were down to ailments such a s heart conditions. General death rates dropped from 1999 to 2006, but sufferers below 65 saw their death rate remain stable – and the over-65 saw theirs increase.

“By 2006, the excess risk in these groups had risk to twice the rate of the general population, whereas prior to that it had only been 1.6 times the risk, so it increased by almost 40%,” said Dr Uy Hoang of Oxford. The study looked at every discharged inpatient with a diagnosis of either condition in England in the relevant time.

Hoang said at the time of the research’s release that doctors should devote attention to predicting and preventing physical illness associated with mental disorders. His study comes at a time when the UK has launched a “no health without mental health” strategy which does attempt to screen for physical illnesses coinciding with mental illnesses. The government aims to reduce the death rate of those with mental disorders.

Rodney Scott described this research result to Wikinews as “Possibly” connected to genetic association with other hereditary ailments, such as cardiovascular disease; he told us another possibility is that “The continued raised mortality rates may be associated with the diseases themselves.”

“We believe the NHS [National Health Service] and Department of Health need to do more to support research and service development for people with bipolar disorder,” Wikinews was told by Suzanne Hudson, Chief Executive of London-based British charity MDF The Bipolar Organisation. “The provision of specialist services for bipolar is very limited in the UK and the demand for our services is unprecedented.”

“A genetic test for bipolar would be a useful tool but the science and ethics are very complex,” Hudson told us, referring to the Nature Genetics genetic study. “Just because someone has ‘bipolar genes’ does not mean they might go on to develop it. Family studies of bipolar show that this is a likely outcome of genetics research in this area. Even if it were possible to accurately predict bipolar in this way, questions about how you treat that person are difficult. For example do you start medication that is not necessary at that point in time?”

“Current treatment is not satisfactory” because it does not always work and has “side effects,” Gejman told us. Robert Whitaker, a US medical journalist and book author, told an audience in New Zealand at the end of August that evidence suggests antidepressant drugs may make children and teenagers worse – “You see many become worse and end up with a more severe diagnosis, like bipolar illness,” and the suicide risk may increase.

Whitaker blames commercial interests. “The adult market appeared saturated, and so they began eying children and teenagers. Prior to this, few children and youth were seen as suffering from major depression, and so few were prescribed anti-depressants.”

One possible alternative, raised by a connection between depressive illness and inflammation, is aspirin and similar compounds. “The link between inflammation and mood disorders has been known for sometime and the use of aspirin and other drugs in depression is now becoming more common in the literature,” Hudson says. “Any new treatments for bipolar, which is a very complex and co-morbid illness, has to be a good thing.”

Professor Dr. Michael Berk, chairman of psychiatry at Australia’s Deakin University, recently gave a talk to just this effect. Speaking at this year’s Congress of the European College of Neuropsychopharmacology, held this past month, he also highlighted statins as a treatment. Recognising the link to physical ailments, he told an interviewer “The brain does not exist in isolation, and we need to understand that pathways similar to those that underpin risks for cardiovascular disorders, stroke, and osteoporosis might also underpin the risk for psychiatric disorders, and that other treatments might be helpful.”

Berk also touched upon speed of diagnosis and treatment; “Early interventions can potentially improve the outcome” of bipolar sufferers, he told his audience. MDF The Bipolar Organisation claim an average of ten years is possible before a person is diagnosed. “This clearly is an issue, if we believe that earlier diagnosis and treatment facilitate better outcomes,” Berk told Wikinews. Though he questions the effectiveness of currently-used drugs on advanced bipolar cases, he does not go so far as to say drugs are actively harmful. He told us “it appears that our best treatments work best earlier in the illness course; and that seems to apply to psychotherapy and pharmacotherapy.”

Berk has already performed research using statins which suggests they can form a treatment. He now seeks funding for research involving aspirin. On funding, he tells Wikinews “psychiatric disorders comprise between 16% and 22% of the burden of disability (depending on who measures it), attracts^[sic] just over 6% of the clinical budget at least in Australia and 3% of the research budget. Research as a discretionary spending item is at great risk.”

Berk’s research, in the past, has been funded by companies including GlaxoSmithKline. Hudson told Wikinews this did not concern her charity; in fact, they welcomed it. “We believe it is important pharmaceutical companies continue to invest in the development of new medications for bipolar. This is how it works in all other health specialities and mental health should be no different.”

“There is a need for greater education for mental health professionals and GPs [general practitioners] about bipolar [in the UK],” she told us. “As the national bipolar charity we receive many, many calls and requests from GPs and other health professionals for our leaflets and information sheets which is fantastic. We very much welcome opportunities to work together for the benefit of individuals affected by bipolar.”

Wikinews contacted the UK’s National Institute for Clinical Excellence (NICE) to discuss issues raised in this article, including future treatments, genetic screening, and mortality rates. NICE did not respond.

Might statins and/or aspirin improve treatment – might they be cheaper, perhaps, or safer? “This is an area of research promise,” says Berk, “however it is too early to make any clinical treatment claims; [all] we can say is that this needs to be studied in properly designed trials capable of giving a more definitive answer.” And what of possible explanations for the increased mortality rate observed in England? Should researchers look at whether bipolar influences more than just the brain, or if it is linked to other genetic conditions?

“For sure,” he told us. “There is new evidence that similar pathways contribute to the risk for both medical and psychiatric illness, both in terms of lifestyle factors, and biomarkers of risk.”

Saturday, January 14, 2006

Israeli Prime Minister Ariel Sharon remains in a coma after 10 days.

Medical experts on the outside say that Sharon’s failure to regain consciousness does not look well for his recovery.

On Wednesday, doctors began reducing the level of sedatives with the intent of drawing Sharon out of an induced coma. Hospital officials say that Sharon remains “lightly sedated”, but are not sure when he will wake up once they stop giving him the sedatives.

New reports say an EEG has shown that Sharon has activity on both sides of his brain and doctors say that’s the most “promising” signs they have had since they started to bring him out of the coma. Doctors also say that activity in both lobes doesn’t indicate anything about the extent of the brain damage. “It is another piece of information that on its own means nothing,” said Dr. Gal Ifergan, a neurologist at Soroka Medical Center in Beer Sheva, who is not treating Sharon.

Although he has activity on both sides of his brain, Sharon could still have extensive brain damage, Ifergan said. He also said that it’s “very worrisome that he hasn’t woken from his coma”, yet.

There are 2 major types of stroke, and Sharon has had both. The first was a mild stroke Dec. 18 caused by a blood clot that lodged in an artery in the brain, cutting off blood flow and causing minor damage. The second occurred just one day before he was scheduled to undergo surgery to repair a hole in his heart. This one was a severe cerebral hemorrhage

“After nine days he has had very slow progress and that indicates more permanent injury to a level where his quality of life is affected,” Dr. Demetrius Lopes, a Rush University Medical Center neurosurgeon said. “And then decisions will have to be made,” he added.

Sharon’s condition is still critical but stable.